Modeling the human p-glycoprotein translocation mechanism using targeted molecular dynamics

Loading...
Thumbnail Image
Author(s)
Speir, Ethan J.
Advisor(s)
Editor(s)
Associated Organization(s)
Organizational Unit
Organizational Unit
School of Biological Sciences
School established in 2016 with the merger of the Schools of Applied Physiology and Biology
Supplementary to:
Abstract
P-glycoprotein (P-gp), a member of the ABC-transporter superfamily, is a transmembrane protein that holds clinical importance due to its role in drug metabolism and multi-drug resistance (MDR) in cancer. Because crystallization attempts have failed at elucidating its 3D structure, recent research efforts have focused on constructing homology models of human P-gp. Prior to 2009, these homology models were primarily based upon bacterial transporters such as Sav1866, MalkK, and MsbA that represented the inward-facing conformation of P-gp. Following the release of an inward-facing mouse P-gp-based homology model of human P-gp, however, simulations modeling the inward to outward transition of P-gp have since been made possible. In this study, a targeted molecular dynamic (TMD) simulation is performed in order to model the translocation mechanism of human P-gp. This simulation provides insight into the gating scheme of P-gp itself as well as other ABC-transporters such as the cystic fibrosis transmembrane conductance regulator (CFTR), a protein implicated in the pathophysiology of cystic fibrosis.
Sponsor
Date
2012-05-07
Extent
Resource Type
Text
Resource Subtype
Undergraduate Thesis
Rights Statement
Rights URI